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DOI: We examined 16 patients with s-JIA and reported the clinical manifestations, laboratory data, treatments and outcomes. We retrospectively reviewed their medical charts.

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Results: There were nine boys and seven girls, with mean age of onset of 7. One patient had Epstein-Barr virus-associated hemophagocytic syndrome complications. Neutrophilic leukocytosis was a common feature. Other laboratory data showed elevated C-reactive protein levels The mean time from onset of symptoms to diagnosis was 9.

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Non-steroidal anti-inflammatory drugs, steroids, disease-modifying anti-rheumatic drugs and anti-tumor necrosis factor agents were used for treatment.

Due to prolonged fever, 2. Most cases had satisfactory therapeutic outcomes except one boy, who had permanent joint contracture. Conclusion: The clinical manifestations of s-JIA in Taiwan were often accompanied by a prolonged fever.

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This results in clinicians often suspecting bacterial infections and prescribing several kinds of antibiotics.

In the case of prolonged fever, s-JIA should always arthritis symptoms causes placed on the list of differential diagnoses.

Copyright © Taiwan Society of Microbiology. Published by Elsevier B. All rights reserved.

University of Edinburgh Summary: Scientists have shed light on why some people are more susceptible to gout than others. A study has identified 18 new genetic variations that increase levels of uric acid in the blood, which is the main cause of the disease. High levels of uric acid form small crystals in joints and tissues, causing pain and swelling -- the main symptoms of the condition once known as the 'disease of kings'. Understanding how these common genetic variants increase uric acid levels in the blood may arthritis symptoms causes to improvements in the treatment and prevention of the disease. It could also help research into urate-lowering drugs.

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